Springer Nature's large fully open-access multidisciplinary journal, carrying a high volume of cancer genomics, immunology, AI and translational papers.
Nature Communications (Nat Commun) is Springer Nature's large, fully open-access multidisciplinary journal, founded in 2010, publishing under CC BY licences with peer-review files often published alongside the article. It publishes thousands of articles a year across science, and in cancer it is a major venue for genomic and single-cell studies, immunology, AI and computational methods, and secondary analyses of trial biospecimens. Its readers are researchers across disciplines who want open-access primary research. Within OnCo it is linked from the biographies of Woong-Yang Park, Robert Gatenby, Giovanni Blandino and Viktor Adalsteinsson, and a reader would use it for the high volume of cancer genomics and translational papers it carries.
One of the most cited papers Europe PMC returns for Santosh Kesari at Asthra Health, so it is a natural starting point for reading their work. The record was linked automatically from the author list and affiliation; read the abstract above and the paper itself before relying on any figure.
This is the paper Europe PMC returns for registry id NCT04956640 with the most citations, so it is the natural first reading for anyone following the trial. Read the abstract above alongside the trial page and the registry entry; the record was linked automatically and its figures have not been checked by hand.
A second publication from the MOUNTAINEER trial, later than the first and described in its title as an update or longer-term analysis. Read it with the primary publication linked from the trial page; the record was linked automatically and its figures have not been checked by hand.
This is the paper Europe PMC returns for registry id NCT05077449 with the most citations, so it is the natural first reading for anyone following the trial. Read the abstract above alongside the trial page and the registry entry; the record was linked automatically and its figures have not been checked by hand.
This is the paper Europe PMC returns for registry id NCT05724563 with the most citations, so it is the natural first reading for anyone following the trial. Read the abstract above alongside the trial page and the registry entry; the record was linked automatically and its figures have not been checked by hand.
One technology page on OnCo cites this paper by its DOI; this record gives the citation a page of its own so a reader can follow it without leaving OnCo. Read the abstract above alongside the citing page listed under Related; the record was created automatically from the Europe PMC entry and its figures have not been checked by hand.
One technology page and one idea page on OnCo cite this paper by its DOI; this record gives the citation a page of its own so a reader can follow it without leaving OnCo. Read the abstract above alongside the citing pages listed under Related; the record was created automatically from the Europe PMC entry and its figures have not been checked by hand.
Molecular confirmation that the visible precursor is not always the parent of the cancer beside it, which tempers hopes that finding and removing dysplasia catches every tumour, and puts Wnt signalling through CTNNB1 at the start of the raised route.
It gives the PD-L1-negative mesenchymal subtype, the group immunotherapy leaves behind, a mechanistic route back to immune visibility, and explains why immune infiltration and subtype are coupled.
One technology page on OnCo cites this paper by its DOI; this record gives the citation a page of its own so a reader can follow it without leaving OnCo. Read the abstract above alongside the citing page listed under Related; the record was created automatically from the Europe PMC entry and its figures have not been checked by hand.
One technology page on OnCo cites this paper by its DOI; this record gives the citation a page of its own so a reader can follow it without leaving OnCo. Read the abstract above alongside the citing page listed under Related; the record was created automatically from the Europe PMC entry and its figures have not been checked by hand.
It gives surveillance a target and a timetable: catch high-grade dysplasia and there are about three years before invasion, and TGF-beta pathway loss is the event to detect.
A three-continent cohort from high-incidence countries, and the origin of the idea that a shared frameshift (ELF3) could be a vaccine target in a cancer with few targets. CTNNB1 and STK11 from this list recur in the MSK data.
One bottleneck page on OnCo cites this paper by its DOI; this record gives the citation a page of its own so a reader can follow it without leaving OnCo. Read the abstract above alongside the citing page listed under Related; the record was created automatically from the Europe PMC entry and its figures have not been checked by hand.
Mucinous ovarian carcinoma is confirmed as a primary ovarian disease with its own biology; HER2 amplification in about a fifth of cases is a possible treatment target.
It supplies the biological reason for treating a BRCA2 carrier's localised disease aggressively rather than watching it, and it settles a long-standing pathology question by showing that intraductal carcinoma and the adjacent invasive tumour are the same clone rather than two separate processes.
One technology page, one pathway page and one term page on OnCo cite this paper by its DOI; this record gives the citation a page of its own so a reader can follow it without leaving OnCo. Read the abstract above alongside the citing pages listed under Related; the record was created automatically from the Europe PMC entry and its figures have not been checked by hand.
The first demonstration that molecular residual disease can be read in pancreatic cancer and that it moves months ahead of the scan.
The microdissected cohort is the cleanest exome reference for allele-level KRAS and copy-number calls, and it first tied MYC amplification to the squamous, chemotherapy-resistant end of the disease.
It matters for how the test is done. A mismatch repair assay designed around the colorectal mechanism, looking for MLH1 promoter methylation and simple mutations, can miss the prostate mechanism entirely, which is one reason microsatellite instability was under-recognised in this disease for so long.
Angelika Eggert is a paediatric oncologist and neuroblastoma researcher.
Eliane Piaggio is an immunologist studying regulatory T cells and immune profiling to guide immunotherapy.
Molecular oncologist known for mutant p53 research, appointed Scientific Director of Rome's Regina Elena National Cancer Institute in 2026.
Cancer pharmacologist at UC San Diego known for work on G-protein signalling and mTOR-targeted therapy in head and neck cancer.
Physician-scientist who is President of Tianjin Medical University Cancer Institute and Hospital, one of China's largest specialist cancer hospitals and a National Clinical Research Center for Malignant Tumors.
Cancer biologist who characterised truncated HER2 (p95HER2) and develops p95HER2-targeted T-cell engagers.
Systems biologist heading research at EMBL-EBI, known for computational methods to model signalling networks and predict cancer drug response.
Thoracic medical oncologist who serves as CEO of the International Association for the Study of Lung Cancer.
Lukas B. Been represents University Medical Center Groningen Comprehensive Cancer Center in the Organisation of European Cancer Institutes, which lists the centre among its members in The Netherlands.
Hungarian oncologist who is Director-General of the National Institute of Oncology in Budapest, Hungary's national cancer centre.
Cancer biologist who leads the Olivia Newton-John Cancer Research Institute, the research arm of the Olivia Newton-John Cancer Wellness and Research Centre in Melbourne.
Molecular oncologist who characterised PML-RARα in acute promyelocytic leukaemia and studies cancer stem cells.
Director of the Kaiser Permanente Division of Research, one of the largest health-system research units in the United States.
Applies evolutionary game theory to cancer treatment, dosing drugs to control rather than eradicate resistant clones.
The neuro-oncologist who founded Asthra Health in Santa Monica, a practice giving outside opinions on complex cancers.
Pioneer of DNA methylation analysis whose Garvan laboratory studies how epigenetic changes drive prostate and breast cancer.
Scientist who directs the Cancer Research UK City of London Centre, the multi-institution London hub for cancer biotherapeutics research.
Engineer who showed low-pass sequencing of blood can replace tumour biopsies and built ultra-sensitive MRD tests.
Built the genome institute behind Samsung Medical Center's precision oncology programme and its single-cell studies of tumours.
Genome biologist who chairs UMC Utrecht's Strategic Program Cancer, the umbrella for more than 800 cancer researchers in Utrecht.
One of the most cited papers Europe PMC returns for Santosh Kesari at Asthra Health, so it is a natural starting point for reading their work. The record was linked automatically from the author list and affiliation; read the abstract above and the paper itself before relying on any figure.
This is the paper Europe PMC returns for registry id NCT04956640 with the most citations, so it is the natural first reading for anyone following the trial. Read the abstract above alongside the trial page and the registry entry; the record was linked automatically and its figures have not been checked by hand.
A second publication from the MOUNTAINEER trial, later than the first and described in its title as an update or longer-term analysis. Read it with the primary publication linked from the trial page; the record was linked automatically and its figures have not been checked by hand.
This is the paper Europe PMC returns for registry id NCT05077449 with the most citations, so it is the natural first reading for anyone following the trial. Read the abstract above alongside the trial page and the registry entry; the record was linked automatically and its figures have not been checked by hand.
This is the paper Europe PMC returns for registry id NCT05724563 with the most citations, so it is the natural first reading for anyone following the trial. Read the abstract above alongside the trial page and the registry entry; the record was linked automatically and its figures have not been checked by hand.
One technology page on OnCo cites this paper by its DOI; this record gives the citation a page of its own so a reader can follow it without leaving OnCo. Read the abstract above alongside the citing page listed under Related; the record was created automatically from the Europe PMC entry and its figures have not been checked by hand.
One technology page and one idea page on OnCo cite this paper by its DOI; this record gives the citation a page of its own so a reader can follow it without leaving OnCo. Read the abstract above alongside the citing pages listed under Related; the record was created automatically from the Europe PMC entry and its figures have not been checked by hand.
Molecular confirmation that the visible precursor is not always the parent of the cancer beside it, which tempers hopes that finding and removing dysplasia catches every tumour, and puts Wnt signalling through CTNNB1 at the start of the raised route.
Shares Genomic characterization of co-existing neoplasia and carcinoma lesions reveals distinct evolutionary paths of gallbladder cancer, Integrated genomic analysis reveals mutated ELF3 as a potential gallbladder cancer vaccine candidate, Genomic characterization of malignant progression in neoplastic pancreatic cysts, Whole-exome sequencing of pancreatic cancer defines genetic diversity and therapeutic targets.
Shares Genomic characterization of co-existing neoplasia and carcinoma lesions reveals distinct evolutionary paths of gallbladder cancer, Integrated genomic analysis reveals mutated ELF3 as a potential gallbladder cancer vaccine candidate.